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OCR: n (type I), The first and most common per cent of 1 by excessive and accounting for approximatel totally patients with SIAD, is charac molality. erratic vasopressin secretion unaffected by changes in plas en demonstrate Although neoplastic conditi SIAD show the is of this type of this pattern, many other cau same abnormality. The path erent defect is unknown, but sever roduction of mechanisms are possible. Ec e expected to tuating vasopressin by neoplasms m cause random release. Rapid on) might also nonosmotic stimuli (e.g. hyp of the be responsible. Electrical inst vasopressin, or ther possible neurogenic pathways that co the neurohypophysis itself, i mechanism